Genetic polymorphism of CYP1A2 increases the risk of myocardial infarction

dc.creator	Cornelis, Marilyn C.
dc.creator	El-Sohemy, Ahmed
dc.creator	Campos Núñez, Hannia
dc.date.accessioned	2020-07-01T18:45:48Z
dc.date.available	2020-07-01T18:45:48Z
dc.date.issued	2004
dc.identifier.citation	https://jmg.bmj.com/content/41/10/758
dc.identifier.issn	1468-6244
dc.identifier.uri	https://hdl.handle.net/10669/81247
dc.description.abstract	There is growing evidence that DNA damage caused by mutagens found in tobacco smoke may contribute to the development of coronary heart disease (CHD). In order to bind to DNA many mutagens require metabolic activation by cytochrome P450 (CYP) 1A1 or CYP1A2. The objective of this study was to determine the effects of CYP1A1 and CYP1A2 genotypes on risk of myocardial infarction (MI) and whether smoking interacts with genotype to modify risk.	es_ES
dc.language.iso	en_US	es_ES
dc.source	Journal of Medical Genetics, Vol. 41, No. 10	es_ES
dc.subject	Tabaco	es_ES
dc.subject	infarto de miocardio	es_ES
dc.subject	enfermedad coronaria	es_ES
dc.title	Genetic polymorphism of CYP1A2 increases the risk of myocardial infarction	es_ES
dc.type	artículo original
dc.identifier.doi	10.1136/jmg.2004.022012
dc.description.procedence	UCR::Vicerrectoría de Investigación::Unidades de Investigación::Ciencias Sociales::Centro Centroamericano de Población (CCP)	es_ES

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